Sherris Medical Microbiology, Sixth Edition

Sherris Medical Microbiology, Sixth Edition

Nafees Ahmad

Language: English

Pages: 1008

ISBN: 0071818219

Format: PDF / Kindle (mobi) / ePub


The most dynamic, comprehensive, and student-friendly text on the nature of microorganisms and the fascinating processes they employ in producing infections disease

A Doody's Core Title for 2015!

For more than a quarter-of-a-century, no other text has explained the link between microbiology and human disease states better than Sherris Medical Microbiology. Through a vibrant, engaging approach, this classic gives you a solid grasp of the significance of etiologic agents, the pathogenic processes, epidemiology, and the basis of therapy for infectious diseases.

Part I of Sherris Medical Microbiology opens with a non-technical chapter that explains the nature of infection and the infection agents. The following four chapters provide more detail about the immune response to infection and the prevention, epidemiology, and diagnosis of infectious disease. Parts II through V form the core of the text with chapters on the major viral, bacterial, fungal, and parasitic diseases. Each of these sections opens with chapters on basic biology, pathogenesis, and antimicrobial agents.

Features and Learning Aids:

  • 57 chapters that simply and clearly describe the strains of viruses, bacteria, fungi, and parasites that can bring about infectious diseases
  • Explanations of host-parasite relationship, dynamics of infection, and host response
  • A clinical cases with USMLE-style questions concludes each chapter on the major viral, bacterial, fungal, and parasitic diseases
  • All tables, photographs, and illustrations are in full color
  • Clinical Capsules cover the essence of the disease(s) caused by major pathogens
  • Margin Notes highlight key points within a paragraph to facilitate review
  • In addition to the chapter-ending case questions, a collection of 100 practice questions is also included

Sometime in the future, an improved understanding of current worldwide infectious disease scourges will lead to their control. Hopefully, you will find the basis for that understanding presented in the pages of this book.

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risk of hepatitis B infection in medical personnel. Attack rates are also high in the sexual partners of infected patients. About 50% of HBV infections in the United States are sexually transmitted Needlestick transmission is a risk for healthcare workers Hepatitis B infection of infants does not appear to be transplacentally transmitted to the fetus in utero, but is acquired during the birth process by the swallowing of infected blood or fluids or through abrasions. The rate of virus

transmission suspected Latency is common, reactivation upon immune suppression Disease associated with immunocompromised patients PATHOGENESIS Polyomaviruses can produce malignant tumors in certain experimental animals, but not in their natural hosts. For example, SV40 can produce lymphocytic leukemia and a variety of reticuloendothelial cell sarcomas in baby hamsters, but is not oncogenic in its natural monkey host. Fortunately, even though it can transform some human cells in vitro, SV40

ankles and then spread up the extremities to the trunk in a few hours. A diagnostic feature of RMSF is the frequent appearance of the rash on the palms and soles, a finding not usually seen in maculopapular eruptions associated with other infections, including typhus. Muscle tenderness, especially in the gastrocnemius, is characteristic and maybe extreme. If untreated, or occasionally in patients despite therapy, complications such as disseminated intravascular coagulation, thrombocytopenia,

suggests a toxic effect, direct evidence is lacking. Several fungi do produce exotoxins, called mycotoxins, in the environment but not in vivo. The structural components of the cell do not cause effects similar to those of the endotoxin of Gram-negative bacteria, although mannan is known to circulate widely in the body. The circulating products of Cryptococcus neoformans have been shown to downregulate immune functions. The injury caused by fungal infections seems to be due primarily to the

373 stationary phase, 373 humans and, 392 immune system and, 397 injury, 400–403 innate defenses against, 394t innate immunity, 397–398 invasion, 396–397, 397f media for isolation of, 81 metabolism, 363–378, 364f simplicity, 364 speed, 364 uniqueness, 364 versatility, 364 microaerophilic, 367 motility, 377–378 mutations, 378–379 nucleoid, 353, 362 pathogenicity, 66 persistence of, 397–400 plasma membrane, 359–361 plasmids, 362 polymerization reactions, 368–371 prokaryotic

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